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Routes of Neuroinflammation Autophagy and Calcium Dependent Mechanisms in Traumatic Brain Injury

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  • Rajiv Kumar

    (University of Delhi, New Delhi, India)

Abstract

Neuroinflammation, immunosuppression, autophagy, and calcium-dependent mechanisms, are those cellular events, which occurred at the site of injury. Prolonged and delayed neuroinflammation promote macrophages and as a result, the activation of resident microglia cells comes into existence. The chain of these biological transformations enhances astrogliosis. The improper connected axonal and increased axonal transport proteins in the node originated prolonged swelling of injured axons and induced apoptotic cell death of neurons [1].

Suggested Citation

  • Rajiv Kumar, 2024. "Routes of Neuroinflammation Autophagy and Calcium Dependent Mechanisms in Traumatic Brain Injury," Biomedical Journal of Scientific & Technical Research, Biomedical Research Network+, LLC, vol. 54(5), pages 46510-46512, February.
    • Handle: RePEc:abf:journl:v:54:y:2024:i:5:p:46510-46512
    DOI: 10.26717/BJSTR.2024.54.008629
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