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piRNA-mediated regulation of transposon alternative splicing in the soma and germ line

Author

Listed:
  • Felipe Karam Teixeira

    (Howard Hughes Medical Institute (HHMI) and Kimmel Center for Biology and Medicine of the Skirball Institute, New York University School of Medicine
    Department of Genetics, University of Cambridge)

  • Martyna Okuniewska

    (Howard Hughes Medical Institute (HHMI) and Kimmel Center for Biology and Medicine of the Skirball Institute, New York University School of Medicine)

  • Colin D. Malone

    (Howard Hughes Medical Institute (HHMI) and Kimmel Center for Biology and Medicine of the Skirball Institute, New York University School of Medicine
    Institute for Genomic Medicine, Columbia University Medical Center)

  • Rémi-Xavier Coux

    (Howard Hughes Medical Institute (HHMI) and Kimmel Center for Biology and Medicine of the Skirball Institute, New York University School of Medicine)

  • Donald C. Rio

    (Center for RNA Systems Biology, and California Institute for Quantitative Biosciences, University of California)

  • Ruth Lehmann

    (Howard Hughes Medical Institute (HHMI) and Kimmel Center for Biology and Medicine of the Skirball Institute, New York University School of Medicine)

Abstract

A new mechanism of pre-mRNA splicing regulation is revealed that is mediated by piRNA pathway components and is dependent on heterochromatin histone modifications.

Suggested Citation

  • Felipe Karam Teixeira & Martyna Okuniewska & Colin D. Malone & Rémi-Xavier Coux & Donald C. Rio & Ruth Lehmann, 2017. "piRNA-mediated regulation of transposon alternative splicing in the soma and germ line," Nature, Nature, vol. 552(7684), pages 268-272, December.
    • Handle: RePEc:nat:nature:v:552:y:2017:i:7684:d:10.1038_nature25018
    DOI: 10.1038/nature25018
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    Cited by:

    1. Jasper Lopik & Azad Alizada & Maria-Anna Trapotsi & Gregory J. Hannon & Susanne Bornelöv & Benjamin Czech Nicholson, 2023. "Unistrand piRNA clusters are an evolutionarily conserved mechanism to suppress endogenous retroviruses across the Drosophila genus," Nature Communications, Nature, vol. 14(1), pages 1-17, December.

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