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Regulation of DNA repair pathway choice in S and G2 phases by the NHEJ inhibitor CYREN

Author

Listed:
  • Nausica Arnoult

    (The Salk Institute for Biological Studies)

  • Adriana Correia

    (The Salk Institute for Biological Studies)

  • Jiao Ma

    (The Salk Institute for Biological Studies)

  • Anna Merlo

    (The Salk Institute for Biological Studies)

  • Sara Garcia-Gomez

    (Dsb Repair Metabolism Laboratory, The Francis Crick Institute)

  • Marija Maric

    (Dsb Repair Metabolism Laboratory, The Francis Crick Institute)

  • Marco Tognetti

    (The Salk Institute for Biological Studies
    Institute of Biochemistry)

  • Christopher W. Benner

    (University of California San Diego)

  • Simon J. Boulton

    (Dsb Repair Metabolism Laboratory, The Francis Crick Institute)

  • Alan Saghatelian

    (The Salk Institute for Biological Studies)

  • Jan Karlseder

    (The Salk Institute for Biological Studies)

Abstract

CYREN is a direct inhibitor of classical non-homologous end joining that promotes error-free repair by homologous recombination during the S and G2 phases of the cell cycle.

Suggested Citation

  • Nausica Arnoult & Adriana Correia & Jiao Ma & Anna Merlo & Sara Garcia-Gomez & Marija Maric & Marco Tognetti & Christopher W. Benner & Simon J. Boulton & Alan Saghatelian & Jan Karlseder, 2017. "Regulation of DNA repair pathway choice in S and G2 phases by the NHEJ inhibitor CYREN," Nature, Nature, vol. 549(7673), pages 548-552, September.
    • Handle: RePEc:nat:nature:v:549:y:2017:i:7673:d:10.1038_nature24023
    DOI: 10.1038/nature24023
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