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Gore et al. reply

Author

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  • Aniket V. Gore

    (Vertebrate Development Group, Temasek Life Sciences Laboratory, 1 Research Link, National University of Singapore
    †Present addresses: Unit on Vertebrate Organogenesis, Laboratory of Molecular Genetics, The National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland 20892, USA (A.V.G.); Cell Culture Process Development, Lonza Biologics, 228 Bath Road, Slough, Berkshire, SL1 4DX, UK (A.C.))

  • Albert Cheong

    (Vertebrate Development Group, Temasek Life Sciences Laboratory, 1 Research Link, National University of Singapore
    †Present addresses: Unit on Vertebrate Organogenesis, Laboratory of Molecular Genetics, The National Institute of Child Health and Human Development, National Institutes of Health, Bethesda, Maryland 20892, USA (A.V.G.); Cell Culture Process Development, Lonza Biologics, 228 Bath Road, Slough, Berkshire, SL1 4DX, UK (A.C.))

  • Patrick C. Gilligan

    (Vertebrate Development Group, Temasek Life Sciences Laboratory, 1 Research Link, National University of Singapore)

  • Karuna Sampath

    (Vertebrate Development Group, Temasek Life Sciences Laboratory, 1 Research Link, National University of Singapore
    † School of Biological Sciences, Nanyang Technological University, 60 Nanyang Drive, 637551 Singapore
    National University of Singapore)

Abstract

Replying to: J. T. Bennett et al. Nature 450, doi:10.1038/nature06314 (2007) We presented several lines of evidence indicating that dorso–ventral asymmetry is apparent in zebrafish embryos by cleavage stages1, one of which showed that injection of three different morpholino oligonucleotides targeting three different squint (sqt) sequences cause severe disruption in dorsal structures. We concluded that the dorsal axis is evident by the 4-cell stage, and suggested that maternal Sqt and associated factors may function in zebrafish dorsal-axis formation1. Bennett et al. challenge our results obtained with morpholino oligonucleotides because they do not find a comparable defect in maternal and zygotic sqt mutants2.

Suggested Citation

  • Aniket V. Gore & Albert Cheong & Patrick C. Gilligan & Karuna Sampath, 2007. "Gore et al. reply," Nature, Nature, vol. 450(7167), pages 2-4, November.
    • Handle: RePEc:nat:nature:v:450:y:2007:i:7167:d:10.1038_nature06315
    DOI: 10.1038/nature06315
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