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Transcription factors FOXG1 and Groucho/TLE promote glioblastoma growth

Author

Listed:
  • Federica Verginelli

    (Centre for Neuronal Survival, Montreal Neurological Institute and Hospital, McGill University)

  • Alessandro Perin

    (Centre for Neuronal Survival, Montreal Neurological Institute and Hospital, McGill University
    Present address: Fondazione IRCCS Istituto Neurologico ‘Carlo Besta’, Neurosurgery Department, Milano, Italy)

  • Rola Dali

    (Centre for Neuronal Survival, Montreal Neurological Institute and Hospital, McGill University)

  • Karen H. Fung

    (Centre for Neuronal Survival, Montreal Neurological Institute and Hospital, McGill University)

  • Rita Lo

    (Centre for Neuronal Survival, Montreal Neurological Institute and Hospital, McGill University)

  • Pierluigi Longatti

    (Treviso Hospital, University of Padova)

  • Marie-Christine Guiot

    (Montreal Neurological Institute and Hospital, McGill University)

  • Rolando F. Del Maestro

    (Brain Tumour Research Centre, Montreal Neurological Institute and Hospital, McGill University)

  • Sabrina Rossi

    (Treviso Hospital, University of Padova)

  • Umberto di Porzio

    (Institute of Genetics and Biophysics, Adriano Buzzati Traverso)

  • Owen Stechishin

    (Hotchkiss Brain Institute, University of Calgary)

  • Samuel Weiss

    (Hotchkiss Brain Institute, University of Calgary)

  • Stefano Stifani

    (Centre for Neuronal Survival, Montreal Neurological Institute and Hospital, McGill University)

Abstract

Glioblastoma (GBM) is the most common and deadly malignant brain cancer, with a median survival of

Suggested Citation

  • Federica Verginelli & Alessandro Perin & Rola Dali & Karen H. Fung & Rita Lo & Pierluigi Longatti & Marie-Christine Guiot & Rolando F. Del Maestro & Sabrina Rossi & Umberto di Porzio & Owen Stechishin, 2013. "Transcription factors FOXG1 and Groucho/TLE promote glioblastoma growth," Nature Communications, Nature, vol. 4(1), pages 1-16, December.
    • Handle: RePEc:nat:natcom:v:4:y:2013:i:1:d:10.1038_ncomms3956
    DOI: 10.1038/ncomms3956
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