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Role of stem-like cells in chemotherapy resistance and relapse in pediatric T-cell acute lymphoblastic leukemia

Author

Listed:
  • Julia Costea

    (EMBL and Medical Faculty of Heidelberg University
    Genome Biology Unit
    Heidelberg University)

  • Kerstin K. Rauwolf

    (University Children’s Hospital)

  • Pietro Zafferani

    (Genome Biology Unit
    Heidelberg University and German Cancer Research Center (DKFZ))

  • Tobias Rausch

    (EMBL and Medical Faculty of Heidelberg University
    Genome Biology Unit
    Genomics Core Facility)

  • Anna Mathioudaki

    (EMBL and Medical Faculty of Heidelberg University
    Molecular Systems Biology Unit
    German Cancer Research Center (DKFZ))

  • Judith Zaugg

    (EMBL and Medical Faculty of Heidelberg University
    Molecular Systems Biology Unit)

  • Martin Schrappe

    (Campus Kiel)

  • Cornelia Eckert

    (Charité Universitätsmedizin Berlin
    German Cancer Research Center (DKFZ))

  • Gabriele Escherich

    (University Medical Center Hamburg-Eppendorf)

  • Jean P. Bourquin

    (University Children’s Hospital)

  • Beat Bornhauser

    (University Children’s Hospital)

  • Andreas E. Kulozik

    (EMBL and Medical Faculty of Heidelberg University
    Heidelberg University
    Hopp Children’s Cancer Center (KiTZ) Heidelberg
    German Cancer Research Center (DKFZ))

  • Jan O. Korbel

    (EMBL and Medical Faculty of Heidelberg University
    Genome Biology Unit
    German Cancer Research Center (DKFZ))

Abstract

T-ALL relapses are characterized by chemotherapy resistance, cellular diversity and dismal outcome. To gain a deeper understanding of the mechanisms underlying relapses, we conduct single-cell RNA sequencing on 13 matched pediatric T-ALL patient-derived samples at diagnosis and relapse, along with samples derived from 5 non-relapsing patients collected at diagnosis. This comprehensive longitudinal single-cell study in T-ALL reveals significant transcriptomic diversity. Notably, 11 out of 18 samples exhibit a subpopulation of T-ALL cells with stem-like features characterized by a common set of active regulons, expression patterns and splice isoforms. This subpopulation, accounting for a small proportion of leukemia cells at diagnosis, expands substantially at relapse, indicating resistance to therapy. Strikingly, increased stemness at diagnosis is associated with higher risk of treatment induction failure. Chemotherapy resistance is validated through in-vitro and in-vivo drug testing. Thus, we report the discovery of treatment-resistant stem-like cells in T-ALL, underscoring the potential for devising future therapeutic strategies targeting stemness-related pathways.

Suggested Citation

  • Julia Costea & Kerstin K. Rauwolf & Pietro Zafferani & Tobias Rausch & Anna Mathioudaki & Judith Zaugg & Martin Schrappe & Cornelia Eckert & Gabriele Escherich & Jean P. Bourquin & Beat Bornhauser & A, 2025. "Role of stem-like cells in chemotherapy resistance and relapse in pediatric T-cell acute lymphoblastic leukemia," Nature Communications, Nature, vol. 16(1), pages 1-14, December.
  • Handle: RePEc:nat:natcom:v:16:y:2025:i:1:d:10.1038_s41467-025-61222-1
    DOI: 10.1038/s41467-025-61222-1
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    References listed on IDEAS

    as
    1. Charles G. Mullighan & Jinghui Zhang & Lawryn H. Kasper & Stephanie Lerach & Debbie Payne-Turner & Letha A. Phillips & Sue L. Heatley & Linda Holmfeldt & J. Racquel Collins-Underwood & Jing Ma & Kenne, 2011. "CREBBP mutations in relapsed acute lymphoblastic leukaemia," Nature, Nature, vol. 471(7337), pages 235-239, March.
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