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Retraction Note: Polymerase θ inhibition activates the cGAS-STING pathway and cooperates with immune checkpoint blockade in models of BRCA-deficient cancer

Author

Listed:
  • Jeffrey Patterson-Fortin

    (Dana-Farber Cancer Institute
    Brigham and Women’s Hospital and Harvard Medical School)

  • Heta Jadhav

    (Dana-Farber Cancer Institute)

  • Constantia Pantelidou

    (Dana-Farber Cancer Institute
    Bayer Pharmaceuticals)

  • Tin Phan

    (Dana-Farber Cancer Institute and Harvard Medical School)

  • Carter Grochala

    (Dana-Farber Cancer Institute and Harvard Medical School
    Arpeggio)

  • Anita K. Mehta

    (Brigham and Women’s Hospital
    Sanofi)

  • Jennifer L. Guerriero

    (Brigham and Women’s Hospital)

  • Gerburg M. Wulf

    (Beth Israel Deaconess Medical Center and Harvard Medical School)

  • Brian M. Wolpin

    (Dana-Farber Cancer Institute
    Brigham and Women’s Hospital and Harvard Medical School
    Dana-Farber Cancer Institute)

  • Ben Z. Stanger

    (University of Pennsylvania Perelman School of Medicine)

  • Andrew J. Aguirre

    (Dana-Farber Cancer Institute
    Brigham and Women’s Hospital and Harvard Medical School
    Dana-Farber Cancer Institute)

  • James M. Cleary

    (Dana-Farber Cancer Institute
    Brigham and Women’s Hospital and Harvard Medical School
    Dana-Farber Cancer Institute)

  • Alan D. D’Andrea

    (Dana-Farber Cancer Institute and Harvard Medical School
    Dana-Farber Cancer Institute)

  • Geoffrey I. Shapiro

    (Dana-Farber Cancer Institute
    Brigham and Women’s Hospital and Harvard Medical School
    Dana-Farber Cancer Institute)

Abstract

No abstract is available for this item.

Suggested Citation

  • Jeffrey Patterson-Fortin & Heta Jadhav & Constantia Pantelidou & Tin Phan & Carter Grochala & Anita K. Mehta & Jennifer L. Guerriero & Gerburg M. Wulf & Brian M. Wolpin & Ben Z. Stanger & Andrew J. Ag, 2023. "Retraction Note: Polymerase θ inhibition activates the cGAS-STING pathway and cooperates with immune checkpoint blockade in models of BRCA-deficient cancer," Nature Communications, Nature, vol. 14(1), pages 1-1, December.
    • Handle: RePEc:nat:natcom:v:14:y:2023:i:1:d:10.1038_s41467-023-43803-0
    DOI: 10.1038/s41467-023-43803-0
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