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Targeting adhesion to the vascular niche to improve therapy for acute myeloid leukemia

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  • Myriam L. R. Haltalli

    (Imperial College London
    The Francis Crick Institute
    University of Cambridge, Jeffrey Cheah Biomedical Centre, Cambridge Biomedical Campus)

  • Cristina Lo Celso

    (Imperial College London
    The Francis Crick Institute)

Abstract

Niche hijack by malignant cells is considered to be a prominent cause of disease relapse. Barbier and colleagues uncover (E)-selectin as a novel mediator of malignant cell survival and regeneration which, upon blockade, has the potential to significantly improve therapeutic outcomes.

Suggested Citation

  • Myriam L. R. Haltalli & Cristina Lo Celso, 2020. "Targeting adhesion to the vascular niche to improve therapy for acute myeloid leukemia," Nature Communications, Nature, vol. 11(1), pages 1-3, December.
    • Handle: RePEc:nat:natcom:v:11:y:2020:i:1:d:10.1038_s41467-020-17594-7
    DOI: 10.1038/s41467-020-17594-7
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