Author
Abstract
In 2018, it was proposed by Reginald B. Little (RBL) that Human Immunodeficiency Virus (HIV) fractionates some isotopes during its infection, dormant stage and advancement to AIDS. In 2023, scientists led by Balter and Chavatte measured fractionation of Zn isotopes during HIV infection and advancement of the disease, whereby the HIV and the HIV infected cells enrich in the least massive stable Zn isotope: 64Zn. The surrounding media of the uninfected cells enriches in the heavier stable isotopes: 66Zn, 67Zn, 68Zn and 70Zn. The author (RBL) previously proposed general mechanism of feeding viruses particular enriched isotopes and the use of electromagnetic fields, magnetic fields, electric fields and agitations to induce isotopic replacements during proliferation of the virus and infection to mutate and inactivate the virus. This work outlines details of the application of such for intrinsic light isotopic enrichment of unstable 62Zn into HIV and HIV infected cells with the rapid electron capture reactions transmuting the 62Zn to 62Cu and to 62Ni to alter interactions of zinc fingers and other binding of zinc in HIV and HIV infected viruses to kill the HIV infected virus and inactivate the HIV with specific emphasis on previously hidden HIV reservoirs. These hidden HIV reservoirs contain Zn fingers and this theoretical cure internally mutates and inactivates these hidden HIV reservoirs throughout the patients’ bodies by nuclei of 62Zn, 62Cu and 62Ni. This work further proposes additional treatment of HIV infected host with excessing 64Zn in the HIV and HIV infected cells with external rotating magnetic waves of electromagnetic radiation (including X-rays, Gamma Rays and Radio Waves), strong static magnetic fields, positrons and neutrinos and antineutrinos for positron induced selective mutations of 64Zn to inactivate the HIV and kill HIV infected cells for an additional route to cure HIV. The author introduces a new phenomenon whereby positrons interacting with electrons in strong magnetic fields and near nuclei to induce stronger neutrino and antineutrino interactions.
Suggested Citation
Handle:
RePEc:epw:physic:v:8:y:2026:i:1:id:70009
DOI: 10.24018/ejphysics.2026.8.1.70009
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