IDEAS home Printed from https://ideas.repec.org/a/epw/ejdent/v4y2023i2id13235.html

Management of Denture-induced Hyperplasia

Author

Listed:
  • Khadija El Assraoui

    (Hassan II University, Morocco)

  • Aicha Oubbaih

    (Hassan II University, Morocco)

  • Khadija Kaoun

    (Hassan II University, Morocco)

  • Samira Bellemkhannate

    (Hassan II University, Morocco)

Abstract

Denture-induced hyperplasia is a reactionary lesion to ill-fittingdentures. It is mostly asymptomatic, but the patient may present pain in case of ulceration. Its etiology is multifactorial. Its main cause is chronic trauma; usually due to prosthetic instability.Its diagnosis is based on clinical examination completed by anatomopathological examination for very extensive lesions. The management of denture-induced hyperplasia depends on the clinical aspect and the size of the lesion. It is based on conditioning alone or associated with the surgical removal of the lesion.Tissue conditioning improves the quality of the prosthetic bearing surfaces and restores the physiological characteristics of the tissues. However, surgical exeresis is indicated in the case of fibrous, old hyperplasia andof an important size where tissue conditioning alone is insufficient. The patient should be informed about the importance of regular control visits to prevent the appearance of hyperplasia. This work aims to compare the management of two cases, with tissue conditioning only for the first, and the combination of the two approaches: surgical removal followed by tissue conditioning for the second.

Suggested Citation

  • Khadija El Assraoui & Aicha Oubbaih & Khadija Kaoun & Samira Bellemkhannate, 2023. "Management of Denture-induced Hyperplasia," European Journal of Dental and Oral Health, European Open Science, vol. 4(2), pages 23-26, March.
    • Handle: RePEc:epw:ejdent:v:4:y:2023:i:2:id:13235
    DOI: 10.24018/ejdent.2023.4.2.235
    as

    Download full text from publisher

    File URL: https://eu-opensci.org/index.php/ejdent/article/view/13235
    File Function: Abstract page
    Download Restriction: no
    File URL: https://eu-opensci.org/index.php/ejdent/article/download/13235/2837
    File Function: Full text
    Download Restriction: no
    File URL: https://libkey.io/10.24018/ejdent.2023.4.2.235?utm_source=ideas
    LibKey link: if access is restricted and if your library uses this service, LibKey will redirect you to where you can use your library subscription to access this item
    ---><---

    More about this item

    Keywords

    ;
    ;
    ;

    Statistics

    Access and download statistics

    Corrections

    All material on this site has been provided by the respective publishers and authors. You can help correct errors and omissions. When requesting a correction, please mention this item's handle: RePEc:epw:ejdent:v:4:y:2023:i:2:id:13235. See general information about how to correct material in RePEc.

    If you have authored this item and are not yet registered with RePEc, we encourage you to do it here. This allows to link your profile to this item. It also allows you to accept potential citations to this item that we are uncertain about.

    We have no bibliographic references for this item. You can help adding them by using this form .

    If you know of missing items citing this one, you can help us creating those links by adding the relevant references in the same way as above, for each refering item. If you are a registered author of this item, you may also want to check the "citations" tab in your RePEc Author Service profile, as there may be some citations waiting for confirmation.

    For technical questions regarding this item, or to correct its authors, title, abstract, bibliographic or download information, contact: Support Team (email available below). General contact details of provider: https://eu-opensci.org/index.php/ejdent .

    Please note that corrections may take a couple of weeks to filter through the various RePEc services.

    IDEAS is a RePEc service. RePEc uses bibliographic data supplied by the respective publishers.